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The harm and medication of hypertensive nephropathy

2017-10-10 11:36

In our daily life, hypertensive nephropathy is the most common symptoms, its own conditions, including kidney disease, causing this pathological change called hypertensive nephropathy. Chronic disease is a persistent increase in blood pressure that causes ischemia and atrophy of the nephron. With the aggravation of the disease, the formation of atherosclerotic renal arteriosclerosis is formed.

The harm and medication of hypertensive nephropathy

Hypertensive nephropathy can cause pathological changes in hypertension and can lead to atherosclerosis. This disease usually occurs in elderly patients, more men than women. The resulting renal artery obstruction is mostly proximal to the 2cm and occasionally involves the distal or its branches of the artery. Hypertensive nephropathy occurs in the intima of the arteries and forms atherosclerotic plaques. It can spread along the vascular wall and narrow the lumen and destroy the intima. The intima is replaced by a mass of acellular plaques, including fat, calcium deposits, and tissue and thrombi that eat necrotic debris.

High blood pressure caused by kidney disease; high renal blood pressure. The acute changes are mainly caused by the hyperplasia of the intima of the large lobar artery and the small wall of the renal arterioles and the narrowing of the lumen. There is a localized necrosis area around the arterial wall and a large amount of fibrin, which is called necrosis of the type of cellulose.

Antihypertensive drugs should be used in the treatment of hypertensive nephropathy. The pathological changes caused by this disease are fibrous muscle hyperplasia, which is common in young patients, more than women. The renal artery lesions mainly occurred in the middle 1/3 and distal 1/3, and the branches were often involved. The unilateral ones were more frequently seen on the right side. Hypertensive nephropathy occurs in the middle layer of the blood vessels, and the smooth muscle and fibrous tissue simultaneously proliferated. The wall of the artery is concentric thickening, which results in intramural hematoma, and there is a large amount of collagen around the hematoma. Angiography shows smooth or narrowing of the renal artery or its branches.

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